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Extracellular CIRP Induces an Inflammatory Phenotype in Pulmonary Fibroblasts via TLR4

Frontiers in Immunology(2021)10.3389/fimmu.2021.721970Source: DataRank Database

Extracellular CIRP Induces an Inflammatory Phenotype in Pulmonary Fibroblasts via TLR4 is a research paper published in Frontiers in Immunology (2021). On theSindex it has a DataRank of 0. It has been cited 22 times.

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Open Access22 citations

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Enrichment:Funding (4 grants)Impact (FWCI 1.57)OA: goldTopics (85)IDs (PubMed, PMC, OpenAlex)Annotations (3)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (4)

Ezgi Sari ORCID,Max Brenner ORCID,Ping Wang ORCID,Siavash Bolourani ORCID

Live enrichment

PubMed:34367191 PMC:PMC8342891 OpenAlex:https://openalex.org/W3185822981

NHLBI NIH HHS

Grant: R01 HL076179

NIGMS NIH HHS

Grant: R35 GM118337

National Institutes of Health

Grant: 5R35GM118337-02

Novel Approaches to Maintaining Organ Function in Sepsis

National Institutes of Health

Grant: 5R01HL076179-10

IMPROVEMENT OF ORGAN FUNCTION AFTER SEVERE HYPOVOLEMIA

FWCI

1.57

Citation Percentile

0.8%

Citation Trend

2021

2022

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.721970/pdf

publisherhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.721970/pdf

publisherhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.721970/full

journalhttps://doi.org/10.3389/fimmu.2021.721970

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34367191

Fields of Study

Interstitial Lung Diseases and Idiopathic Pulmonary FibrosisInhalation and Respiratory Drug DeliveryPneumonia and Respiratory Infections0301 basic medicine03 medical and health sciences0303 health sciencesAnimalsComputational BiologyCytokinesDisease Models, AnimalDisease SusceptibilityExtracellular SpaceFibroblastsGene ExpressionGene Expression ProfilingInflammationInflammation MediatorsMiceMice, KnockoutPhenotypePulmonary FibrosisRNA-Binding ProteinsSignal TransductionToll-Like Receptor 4

MeSH Terms

AnimalsDisease Models, AnimalDisease SusceptibilityExtracellular SpaceFibroblastsInflammationPhenotypePulmonary FibrosisSignal TransductionGene ExpressionCytokinesRNA-Binding ProteinsMice, KnockoutInflammation MediatorsComputational BiologyGene Expression ProfilingToll-Like Receptor 4Mice

Keywords

Pulmonary fibrosisProinflammatory cytokineBleomycinFibroblastTLR4FibrosisExtracellular matrixInflammationMyofibroblastIdiopathic pulmonary fibrosisBiologyTranscriptomePhenotypeExtracellularCancer researchLungCell biologyGene expressionMedicineImmunologyPathologyGeneIn vitroInternal medicineGeneticsEcirpBleomicynMiceAnimalsMice, KnockoutGene Expression ProfilingComputational BiologyRNA-Binding ProteinsRC581-607FibroblastsToll-Like Receptor 4Disease Models, AnimalCytokinesDisease SusceptibilityImmunologic diseases. AllergyInflammation MediatorsExtracellular SpaceSignal Transduction

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

Extracellular CIRP Induces an Inflammatory Phenotype in Pulmonary Fibroblasts via TLR4

FAIR Checklist

Authors (4)

Funding (4 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)