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Gain-of-function of TRPM4 predisposes mice to psoriasiform dermatitis

Frontiers in Immunology(2022)10.3389/fimmu.2022.1025499Source: DataRank Database

Gain-of-function of TRPM4 predisposes mice to psoriasiform dermatitis is a research paper published in Frontiers in Immunology (2022). On theSindex it has a DataRank of 0. It has been cited 11 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access11 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (2 grants)Impact (FWCI 1.05)OA: goldTopics (53)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (9)

Simon Vu ORCID,Xuesong Wu ORCID,Zhenrui Shi ORCID,Desiree Morris,Joshua Bloomstein ORCID

Live enrichment

PubMed:36341417 PMC:PMC9632438 OpenAlex:https://openalex.org/W4306878911

NINDS NIH HHS

Grant: R01 NS128180

National Institutes of Health

Grant: 1R01NS128180-01

Activation and Inhibition Mechanisms of Calcium-Activated Nonselective Cation Channels

FWCI

1.05

Citation Percentile

0.7%

Citation Trend

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.3389/fimmu.2022.1025499

publisherhttps://doi.org/10.3389/fimmu.2022.1025499

publisherhttps://www.frontiersin.org/articles/10.3389/fimmu.2022.1025499/full

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36341417

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/9632438

Fields of Study

Ion Channels and ReceptorsDermatology and Skin DiseasesAsthma and respiratory diseases0301 basic medicine03 medical and health sciences0303 health sciences

MeSH Terms

Gain of Function MutationImiquimodAnimalsEczemaHumansPsoriasisSkinTRPM Cation ChannelsMice

Keywords

Transient receptor potential channelPsoriasisPhenotypeInflammationMutantImmunologyImiquimodChemistryBiologyCell biologyReceptorCancer researchGeneticsGeneKeratinocyteDendritic CellGlibenclamideMouse ModelTrpm4Imiquimod (Imq)Progressive Symmetric Erythrokeratodermia570Medical PhysiologyEczema610TRPM Cation ChannelsMice2.1 Biological and endogenous factorsHumansAnimalsAetiologySkinBiomedical and Clinical SciencesRC581-607Medical MicrobiologyBiochemistry and cell biologyGain of Function MutationImmunologic diseases. Allergy

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being