Altered Gut Microbiome and Fecal Immune Phenotype in Early Preterm Infants With Leaky Gut is a research paper published in Frontiers in Immunology (2022). On theSindex it has a DataRank of 0. It has been cited 49 times.
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National Institute of Diabetes and Digestive and Kidney Diseases
Grant: R21DK123674
Gerber Foundation
Grant: 6361
National Institute of Allergy and Infectious Diseases
Grant: U19AI145825 , P01AI125181
NIAID NIH HHS
Grant: P01 AI125181
NIAID NIH HHS
Grant: U19 AI145825
NHLBI NIH HHS
Grant: R01 HL148672
National Institutes of Health
Grant: 5P01AI125181-05
Pathogenesis of E. coli and Shigella infections in human enteroid models
National Institutes of Health
Grant: 5R21DK123674-02
Role of short chain fatty acids in intestinal barrier maturation in preterm infants
National Institutes of Health
Grant: 5U19AI145825-02
Maternal Immunization and Determinants of Infant Immunity
FWCI
5.13
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 9 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 9 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 10 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 10 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 8 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 5 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 6 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 7 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 8 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 7 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 4 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 6 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 3 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 4 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 5 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 3 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 1 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 1 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 2 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 2 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery