Methamphetamine signals transcription of IL1β and TNFα in a reactive oxygen species-dependent manner and interacts with HIV-1 Tat to decrease antioxidant defense mechanisms is a research paper published in Frontiers in Cellular Neuroscience (2022). On theSindex it has a DataRank of 0. It has been cited 17 times.
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National Institutes of Health
Grant: NIH R03 DA027936
National Institutes of Health
Grant: R21 DA029491
National Institutes of Health
Grant: R01 DA036164
National Institutes of Health
Grant: 1R21DA029491-01A1
Methamphetamine and Immune Cells in NeuroAIDS
National Institutes of Health
Grant: 1R01DA047822-01
Dopamine system as reporter of HIV status and inflammation in Meth abusers
National Institutes of Health
Grant: 3R01DA036164-04S1
Methamphetamine and HIV interactions in the regulation of glial activation
National Institutes of Health
Grant: 1R03DA027936-01
Free Radicals and Methamphetamine Abuse
NIDA NIH HHS
Grant: R01 DA047822
FWCI
0.94
Citation Percentile
0.8%
Citation Trend
Fields of Study
Keywords
Sustainable Development Goals
Additional file 2 of Change of histone H3 lysine 14 acetylation stoichiometry in human monocyte derived macrophages as determined by MS-based absolute targeted quantitative proteomic approach: HIV infection and methamphetamine exposure
Additional file 2 of Change of histone H3 lysine 14 acetylation stoichiometry in human monocyte derived macrophages as determined by MS-based absolute targeted quantitative proteomic approach: HIV infection and methamphetamine exposure
Additional file 1 of Change of histone H3 lysine 14 acetylation stoichiometry in human monocyte derived macrophages as determined by MS-based absolute targeted quantitative proteomic approach: HIV infection and methamphetamine exposure
Additional file 1 of Change of histone H3 lysine 14 acetylation stoichiometry in human monocyte derived macrophages as determined by MS-based absolute targeted quantitative proteomic approach: HIV infection and methamphetamine exposure