Metal Exposure and SNCA rs356219 Polymorphism Associated With Parkinson Disease and Parkinsonism is a research paper published in Frontiers in Neurology (2020). On theSindex it has a DataRank of 0. It has been cited 26 times.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
FAIR checklist signals are shown for context only and do not affect DataRank scoring.
We only score data papers we can read in full — never from an abstract alone.
Istituto Nazionale per l'Assicurazione Contro Gli Infortuni sul Lavoro
Grant: 60002.02/07/2012
National Institutes of Health
Grant: P30 ES023515
National Institutes of Health
Grant: R01ES019222-06A1
National Institutes of Health
Grant: R56-R01ES019222-06
Sixth Framework Programme
Grant: FOODCT-2006-016253
National Institutes of Health
Grant: 2P30ES023515-05
The Mount Sinai Transdisciplinary Center on Early Environmental Exposures
National Institutes of Health
Grant: 1P30ES023515-01
Phenotyping and Stress Assessment Facility Core (PSAFC)
National Institutes of Health
Grant: 5R01ES019222-04
Neurologic function in children exposed to ambient manganese
FWCI
0.84
Citation Percentile
0.8%
Citation Trend
Fields of Study
Keywords
Sustainable Development Goals
Additional file 6 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 6 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 4 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 4 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 5 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 2 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 3 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 5 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 3 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 1 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 2 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function
Additional file 1 of A partial Drp1 knockout improves autophagy flux independent of mitochondrial function