Kinin B1R Activation Induces Endoplasmic Reticulum Stress in Primary Hypothalamic Neurons is a research paper published in Frontiers in Pharmacology (2022). On theSindex it has a DataRank of 0. It has been cited 13 times.
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NHLBI NIH HHS
Grant: R01 HL153115
National Heart, Lung, and Blood Institute
Grant: 5R01HL153115
National Institutes of Health
Grant: 5R01HL153115-05
Neuroimmune Mechanisms of Kinin B1 Receptor in Hypertension
FWCI
1.40
Citation Percentile
0.8%
Citation Trend
Fields of Study
Keywords
Sustainable Development Goals
Additional file 1 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis
Additional file 1 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis
Additional file 2 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis
Additional file 2 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis
Additional file 3 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis
Additional file 3 of Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis