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The ‘Reverse FDUF’ Mechanism of Atrial Excitation–Contraction Coupling Sustains Calcium Alternans—A Hypothesis

Biomolecules(2022)10.3390/biom13010007Source: DataRank Database

The ‘Reverse FDUF’ Mechanism of Atrial Excitation–Contraction Coupling Sustains Calcium Alternans—A Hypothesis is a research paper published in Biomolecules (2022). On theSindex it has a DataRank of 0. It has been cited 3 times.

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (20 grants)Impact (FWCI 0.46)OA: goldTopics (76)IDs (PubMed, PMC, OpenAlex)SDGs (1)

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Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (2)

Lothar A. Blatter ORCID,Kathrin Banach ORCID

Live enrichment

PubMed:36671392 PMC:PMC9855423 OpenAlex:https://openalex.org/W4313468543

National Institutes of Health

Grant: HL057832

National Institutes of Health

Grant: HL062231

National Institutes of Health

Grant: HL128330

National Institutes of Health

Grant: HL132871

National Institutes of Health

Grant: HL134781

National Institutes of Health

Grant: HL155762

National Institutes of Health

Grant: HL164453

NHLBI NIH HHS

Grant: R01 HL128330

NHLBI NIH HHS

Grant: R01 HL134781

NHLBI NIH HHS

Grant: R01 HL132871

NHLBI NIH HHS

Grant: R01 HL155762

NHLBI NIH HHS

Grant: R01 HL164453

NHLBI NIH HHS

Grant: R01 HL062231

National Institutes of Health

Grant: 5R01HL057832-03

CONTROL OF CA++- INDUCED CA++ RELEASE IN HEART

National Institutes of Health

Grant: 5R01HL155762-04

IP3 receptor, NOX2 and calcium signaling domains in atrial physiology and pathophysiology

National Institutes of Health

Grant: 1R01HL132871-01A1

Pathophysiological Regulation of Atrial Alternans and Atrial Fibrillation

National Institutes of Health

Grant: 7R01HL062231-10

E-C Coupling and Ca2+ Regulation atrial myocytes

National Institutes of Health

Grant: 5R01HL128330-03

The Role of p21-activated Kinase (Pak1) in Atrial Fibrillation

National Institutes of Health

Grant: 5R01HL134781-02

Pathophysiological Regulation of Atrial Myocyte Excitation-Contraction Coupling and Calcium Signaling

National Institutes of Health

Grant: 5R01HL164453-04

Atrial Excitation-Contraction Coupling, Calcium Signaling and Electro-Mechanical Alternans

FWCI

0.46

Citation Percentile

0.7%

Citation Trend

2023

2024

2025

goldLicense: cc-by

journalhttps://www.mdpi.com/2218-273X/13/1/7/pdf?version=1672210887

publisherhttps://www.mdpi.com/2218-273X/13/1/7/pdf?version=1672210887

publisherhttps://www.mdpi.com/2218-273X/13/1/7/pdf

journalhttps://doi.org/10.3390/biom13010007

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36671392

Fields of Study

Cardiac electrophysiology and arrhythmiasIon channel regulation and functionCardiac Arrhythmias and Treatments0301 basic medicine0303 health sciences03 medical and health sciencesAnimalsRabbitsCalciumAtrial FibrillationCalcium SignalingHeart AtriaAction PotentialsMyocytes, Cardiac

MeSH Terms

Action PotentialsAnimalsAtrial FibrillationCalciumHeart AtriaRabbitsCalcium SignalingMyocytes, Cardiac

Keywords

Refractory periodAtrial myocytesMyocyteInternal medicineCalciumBeat (acoustics)Contraction (grammar)Excitation–contraction couplingBiophysicsOptical mappingCardiologyChemistryT wave alternansRyanodine receptorBiologyMedicineSudden cardiac deathPhysicsAction potentialAPDcatSRARsarcoplasmic reticulumEccCytosolic Calcium ConcentrationRyrApAp DurationLccCircCruJunctional SrCa TransientCa Release UnitFduf MechanismAlternans RatioFire-diffuse-uptake-fire MechanismJ-srNj-srNon-junctional SrVoltage-gated L-type Ca Channel[Ca]iCa-induced Ca ReleaseBrief ReportAction PotentialsMicrobiologyQR1-502Atrial FibrillationAnimalsMyocytes, CardiacRabbitsCalcium SignalingHeart Atria

Sustainable Development Goals

SDG 0: Life in Land