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TLR3-Dependent Activation of TLR2 Endogenous Ligands via the MyD88 Signaling Pathway Augments the Innate Immune Response

Cells(2020)10.3390/cells9081910Source: DataRank Database

TLR3-Dependent Activation of TLR2 Endogenous Ligands via the MyD88 Signaling Pathway Augments the Innate Immune Response is a research paper published in Cells (2020). On theSindex it has a DataRank of 0. It has been cited 22 times.

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Open Access22 citations

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Enrichment:Funding (4 grants)Impact (FWCI 0.43)OA: goldTopics (82)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (5)

Jiawei Zhao ORCID,Ethan Kazmierski,Denis F. Kinane ORCID,Manjunatha R. Benakanakere ORCID,Hellen S. Teixeira

Live enrichment

PubMed:32824595 PMC:PMC7464415 OpenAlex:https://openalex.org/W3049299236

National Institute of Dental and Craniofacial Research

Grant: DE017384

School of Dental Medicine, University of Pennsylvania

Grant: Internal funding

NIDCR NIH HHS

Grant: R01 DE024160

National Institutes of Health

Grant: 1R01DE017384-01A2

Epithelial Cell TLRs in Disease Susceptibility

FWCI

0.43

Citation Percentile

0.6%

Citation Trend

2021

2022

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://www.mdpi.com/2073-4409/9/8/1910/pdf?version=1597746687

publisherhttps://www.mdpi.com/2073-4409/9/8/1910/pdf?version=1597746687

publisherhttps://www.mdpi.com/2073-4409/9/8/1910/pdf

journalhttps://doi.org/10.3390/cells9081910

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32824595

Fields of Study

Immune Response and InflammationNF-κB Signaling PathwaysImmune cells in cancer0301 basic medicine03 medical and health sciences0303 health sciencesAnimalsCells, CulturedEpithelial CellsFemaleGingivaHealthy VolunteersHumansImmunity, InnateLigandsMiceMice, Inbred C57BLMice, KnockoutMyeloid Differentiation Factor 88Signal TransductionToll-Like Receptor 2Toll-Like Receptor 3Transfection

MeSH Terms

AnimalsCells, CulturedEpithelial CellsFemaleGingivaHumansImmunity, InnateLigandsMice, Inbred C57BLTransfectionSignal TransductionMice, KnockoutToll-Like Receptor 2Toll-Like Receptor 3MiceMyeloid Differentiation Factor 88Healthy Volunteers

Keywords

TLR3TLR2Downregulation and upregulationInnate immune systemTRIFToll-like receptorCell biologyGene silencingSignal transductionIRF3Signal transducing adaptor proteinReceptorChemistryTLR4BiologyImmune systemImmunologyBiochemistryHsp60Hmgb1Pro-inflammatory CytokineMyD88Human Gingival Epithelial CellsGingivaLigandsTransfectionArticleMiceAnimalsHumansCells, CulturedMice, KnockoutQH573-671Epithelial CellsHealthy VolunteersImmunity, InnateToll-Like Receptor 2Toll-Like Receptor 3Mice, Inbred C57BLMyeloid Differentiation Factor 88FemaleCytology

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

TLR3-Dependent Activation of TLR2 Endogenous Ligands via the MyD88 Signaling Pathway Augments the Innate Immune Response

FAIR Checklist

Authors (5)

Funding (4 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)