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Non-Muscle MLCK Contributes to Endothelial Cell Hyper-Proliferation through the ERK Pathway as a Mechanism for Vascular Remodeling in Pulmonary Hypertension

International Journal of Molecular Sciences(2022)10.3390/ijms232113641Source: DataRank Database

Non-Muscle MLCK Contributes to Endothelial Cell Hyper-Proliferation through the ERK Pathway as a Mechanism for Vascular Remodeling in Pulmonary Hypertension is a research paper published in International Journal of Molecular Sciences (2022). On theSindex it has a DataRank of 0. It has been cited 9 times.

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Open Access9 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (6 grants)Impact (FWCI 1.22)OA: goldTopics (56)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (9)

Janae Gonzales ORCID,Rachel Halstrom,Noman Baig,Cat Humpal,R. Demeritte

Live enrichment

PubMed:36362426 PMC:PMC9654627 OpenAlex:https://openalex.org/W4308581858

National Heart, Lung, and Blood Institute of the National Institutes of Health

Grant: HL133474

National Heart, Lung, and Blood Institute of the National Institutes of Health

Grant: HL144909

NHLBI NIH HHS

Grant: K08 HL133474

NHLBI NIH HHS

Grant: T32 HL144909

National Institutes of Health

Grant: 5K08HL133474-04

The role of NFATc3 in altered hypoxic pulmonary vasoconstriction during acute lung injury

National Institutes of Health

Grant: 1T32HL144909-01A1

Postdoctoral Training Program in Pulmonary and Critical Care Translational Research

FWCI

1.22

Citation Percentile

0.8%

Citation Trend

2023

2024

2025

goldLicense: cc-by

journalhttps://www.mdpi.com/1422-0067/23/21/13641/pdf?version=1667816901

publisherhttps://www.mdpi.com/1422-0067/23/21/13641/pdf?version=1667816901

publisherhttps://www.mdpi.com/1422-0067/23/21/13641/pdf

journalhttps://doi.org/10.3390/ijms232113641

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36362426

Fields of Study

Pulmonary Hypertension Research and TreatmentsBone health and treatmentsProtein Kinase Regulation and GTPase Signaling0301 basic medicine0303 health sciences03 medical and health sciences

MeSH Terms

Pulmonary Arterial HypertensionAnimalsDisease Models, AnimalHumansHypertension, PulmonaryMyosin-Light-Chain KinasePulmonary ArteryMAP Kinase Signaling SystemVascular Endothelial Growth Factor AEndothelial CellsCell ProliferationBone Morphogenetic Protein Receptors, Type IIFamilial Primary Pulmonary HypertensionVascular Remodeling

Keywords

Downregulation and upregulationPathogenesisMAPK/ERK pathwayVascular remodelling in the embryoMyosin light-chain kinaseCancer researchCell biologyVascular smooth musclePulmonary hypertensionBMPR2MedicineBiologySignal transductionBone morphogenetic proteinImmunologyInternal medicineMyosinSmooth muscleBiochemistryCytoskeletonVascular remodelingMyosin Light Chain KinaseEndothelial CellsVascular Endothelial Growth Factor APulmonary Arterial HypertensionMAP Kinase Signaling SystemHypertension, PulmonaryPulmonary ArteryBone Morphogenetic Protein Receptors, Type IIArticleDisease Models, AnimalHumansAnimalsFamilial Primary Pulmonary HypertensionMyosin-Light-Chain KinaseCell Proliferation

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SDG 0: No poverty