Histone deposition pathways determine the chromatin landscapes of H3.1 and H3.3 K27M oncohistones is a research paper published in eLife (2020). On theSindex it has a DataRank of 1.9. It has been cited 72 times, with 58 citing works in its 1-hop citation network.
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Linked data & code
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Base Score Contribution
0.644
From this paper's citation signal
Citation Network Contribution
1.3
From 54 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 58 citers.
National Institutes of Health
Grant: R01GM108699
National Institutes of Health
Grant: T32 CA009351
National Institutes of Health
Grant: 5T32CA009351-27
Pediatric Oncology Research Training Program
National Institutes of Health
Grant: 5R01GM108699-04
Development and architecture of epigenetic regulatory elements
Alex's Lemonade Stand Foundation for Childhood Cancer
Damon Runyon Cancer Research Foundation
Howard Hughes Medical Institute
Howard Hughes Medical Institute
FWCI
3.74
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Histone H3.3 K27M chromatin functions implicate a network of neurodevelopmental factors including ASCL1 and NEUROD1 in DIPG
Additional file 1 of Histone H3.3 K27M chromatin functions implicate a network of neurodevelopmental factors including ASCL1 and NEUROD1 in DIPG
Additional file 1 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies
Additional file 1 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies
Additional file 2 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies
Additional file 2 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies
Additional file 3 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies
Additional file 3 of Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies