🏆 Finalist — NIH Data Sharing Index (“S-Index”) Challenge

Maxwell J. Heinrich

Boston Children's Hospital

ORCID: 0009-0004-2368-3470

Also affiliated with Harvard University, Massachusetts Institute of Technology

Biochemistry, Genetics and Molecular BiologyNeuroscience

Pilot corpus only

This score is computed over theSindex pilot corpus and does not cover the full scientific literature. Scores are relative to papers we have ingested — papers, authors, and institutions outside the pilot are not represented. Methodology.

Top 100%percentile

0Author DataRank

Indexed papers

0in pilot corpus

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Papers

Driven by 3 papers. Top paper: “Non-ionotropic signaling through the NMDA receptor GluN2B carboxy terminal domain drives morphological plasticity of dendritic spines and reverses fragile X phenotypes in mouse hippocampus”.

Non-ionotropic signaling through the NMDA receptor GluN2B carboxy terminal domain drives morphological plasticity of dendritic spines and reverses fragile X phenotypes in mouse hippocampus

bioRxiv (Cold Spring Harbor Laboratory)(2024)10.1101/2024.12.15.628559

N/A

0DataRank · unranked

0 citations

Aurore Thomazeau, Peter S.B. Finnie, Maxwell J. Heinrich, Arnold J. Heynen, Noboru H. Komiyama +5 more

Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes

Cell Reports(2025)10.1016/j.celrep.2025.115311

N/A

0DataRank · unranked

11 citations

Aurore Thomazeau, Peter S.B. Finnie, Maxwell J. Heinrich, Arnold J. Heynen, Noboru H. Komiyama +5 more

Cell-intrinsic mechanisms underlying spontaneous activity in the mouse visual cortical slice: implications for fragile X pathophysiology

Journal of Neurophysiology(2025)10.1152/jn.00241.2025

N/A

0DataRank · unranked

0 citations

Mark F. Bear, Maxwell J. Heinrich