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Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes

Cell Reports(2025)10.1016/j.celrep.2025.115311Source: DataRank Database

Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes is a research paper published in Cell Reports (2025). On theSindex it has a DataRank of 0. It has been cited 11 times.

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Scored on demand from live citation data

Open Access11 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (8 grants)Impact (FWCI 10.45)OA: goldTopics (61)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (10)

Aurore Thomazeau ORCID,Peter S.B. Finnie ORCID,Maxwell J. Heinrich ORCID,Arnold J. Heynen ORCID,Noboru H. Komiyama ORCID

Live enrichment

PubMed:39983718 PMC:PMC12006837 OpenAlex:https://openalex.org/W4407768730

National Institute of Mental Health

Grant: R21NS123499

NINDS NIH HHS

Grant: R21 NS087225

NICHD NIH HHS

Grant: R01 HD046943

NINDS NIH HHS

Grant: P01 NS039321

European Commission

Grant: 892837

Unconventional NMDAR signaling theory for Fragile X Syndrome

National Institutes of Health

Grant: 1R21NS123499-01A1

Pathophysiology and treatment of fragile X and related disorders

FRAXA Research Foundation

National Institutes of Health

FWCI

10.45

Citation Percentile

1.0%

Citation Trend

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1016/j.celrep.2025.115311

publisherhttps://doi.org/10.1016/j.celrep.2025.115311

publisherhttps://api.elsevier.com/content/article/PII:S2211124725000828?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S2211124725000828?httpAccept=text/plain

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39983718

Fields of Study

Genetics and Neurodevelopmental DisordersNeuroscience and Neuropharmacology ResearchMitochondrial Function and Pathology03 medical and health sciences0302 clinical medicine

MeSH Terms

Protein DomainsAnimalsFragile X SyndromeMaleMice, Inbred C57BLNeuronal PlasticityPhenotypeSignal TransductionReceptors, N-Methyl-D-AspartateReceptors, Metabotropic GlutamateMice, KnockoutLong-Term Synaptic DepressionDendritic SpinesMiceFragile X Messenger Ribonucleoprotein 1Fragile X Mental Retardation Protein

Keywords

Ionotropic effectDendritic spinePhenotypeNMDA receptorCell biologyTerminal (telecommunication)ChemistryBiologyNeuroscienceReceptorGeneticsComputer networkComputer scienceGeneAudiogenic seizuresLong-term depressionLTDNMDARFragile X SyndromeEpileptogenesisMglur5Dendritic SpinesFxsAgsGlyx-13Non-ionotropicCp: NeuroscienceMice, KnockoutMaleNeuronal PlasticityFragile X Messenger Ribonucleoprotein 1Long-Term Synaptic DepressionReceptors, Metabotropic GlutamateReceptors, N-Methyl-D-AspartateArticleMice, Inbred C57BLMiceProtein DomainsAnimalsSignal Transduction