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Di-valent siRNA-mediated silencing of MSH3 blocks somatic repeat expansion in mouse models of Huntington’s disease

Molecular Therapy(2023)10.1016/j.ymthe.2023.05.006Source: DataRank Database

Di-valent siRNA-mediated silencing of MSH3 blocks somatic repeat expansion in mouse models of Huntington’s disease is a research paper published in Molecular Therapy (2023). On theSindex it has a DataRank of 0. It has been cited 56 times.

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Open Access56 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (13 grants)Impact (FWCI 6.57)OA: hybridTopics (58)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (21)

Jillian Belgrad ORCID,Chantal Ferguson ORCID,Ashley Summers ORCID,Ellen Sapp ORCID,Cassandra McHugh ORCID

Live enrichment

PubMed:37177784 PMC:PMC10277892 OpenAlex:https://openalex.org/W4376598552

CHDI Foundation

Grant: CHDI-6367

CHDI Foundation

Grant: R35 GM131839

CHDI Foundation

Grant: S10 OD020012

National Institutes of Health

Grant: U01 NS114098

National Institutes of Health

Grant: R01 NS106245

NINDS NIH HHS

Grant: F31 NS132424

NINDS NIH HHS

Grant: R01 NS104022

National Institutes of Health

Grant: 1S10OD020012-01

Mid-Scale RNA Synthesis, Purification and Quality Control System

National Institutes of Health

Grant: 5R35GM131839-05

Chemical engineering of therapeutic RNAs for extrahepatic delivery

National Institutes of Health

Grant: 5U01NS114098-05

Advancing RNA Therapeutics for Huntington’s Disease

National Institutes of Health

Grant: 1R01NS106245-01

Selective Editing of the Mutant Huntingtin Gene

Biogen

Huntington's Disease Society of America

FWCI

6.57

Citation Percentile

1.0%

Citation Trend

2023

2024

2025

2026

hybridLicense: cc-by

journalhttp://www.cell.com/article/S1525001623002629/pdf

publisherhttp://www.cell.com/article/S1525001623002629/pdf

publisherhttps://api.elsevier.com/content/article/PII:S1525001623002629?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S1525001623002629?httpAccept=text/plain

journalhttps://doi.org/10.1016/j.ymthe.2023.05.006

Fields of Study

Genetic Neurodegenerative DiseasesRNA Research and SplicingMuscle Physiology and Disorders03 medical and health sciences0302 clinical medicineAnimalsMiceCorpus StriatumHuntingtin ProteinHuntington DiseaseNeostriatumRNA, Double-StrandedRNA, Small InterferingTrinucleotide Repeat ExpansionMutS Homolog 3 Protein

MeSH Terms

Huntingtin ProteinMutS Homolog 3 ProteinAnimalsCorpus StriatumHuntington DiseaseRNA, Double-StrandedNeostriatumTrinucleotide Repeat ExpansionRNA, Small InterferingMice

Keywords

Gene silencingTrinucleotide repeat expansionSomatic cellHuntington's diseaseBiologyDiseaseHuntingtinGeneticsCell biologyCancer researchVirologyGeneMedicineMutantAlleleMismatch repairNeurodegenerationSiRNAHuntington’s DiseaseDna InstabilityOligonucleotide TherapeuticsCag Expansion DisordersHuntingtin ProteinCorrectionCorpus StriatumNeostriatumMiceHuntington DiseaseMutS Homolog 3 ProteinAnimalsOriginal ArticleRNA, Small InterferingRNA, Double-Stranded

Sustainable Development Goals

SDG 0: Good health and well-being