Reactive astrocytes transduce inflammation in a blood-brain barrier model through a TNF-STAT3 signaling axis and secretion of alpha 1-antichymotrypsin is a research paper published in Nature Communications (2022). On theSindex it has a DataRank of 0. It has been cited 188 times.
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U.S. Department of Health & Human Services | National Institutes of Health
Grant: R01 GM117650
U.S. Department of Health & Human Services | National Institutes of Health
Grant: R21 AG070859
NEI NIH HHS
Grant: P30 EY008126
NINDS NIH HHS
Grant: R01 NS084030
NCATS NIH HHS
Grant: UL1 TR002243
NINDS NIH HHS
Grant: RF1 NS129735
NINDS NIH HHS
Grant: T32 NS115706
NICHD NIH HHS
Grant: P50 HD103537
NIA NIH HHS
Grant: F30 AG066418
National Institutes of Health
Grant: 1F30AG066418-01
Elucidating cellular pathways controlling the reactive state of astrocytes in Alzheimer’s Disease
National Institutes of Health
Grant: 1R21AG070859-01
Microvascular Network Degeneration in a Novel Mouse Model of Alzheimer's Disease and Cerebral Amyloid Angiopathy
National Institutes of Health
Grant: 5P30EY008126-08
CORE--ILLUSTRATION AND PHOTOGRAPHY MODULE
National Institutes of Health
Grant: 3P30CA068485-23S2
Cancer Center Support Grant
National Institutes of Health
Grant: 1G20RR030956-01
VANTAGE:Consolidation to create the Vanderbilt Technologies for Advanced Genomics
National Institutes of Health
Grant: 1RF1NS129735-01
STAT3 activation in astrocytes as a driver of neurovascular dysfunction in Alzheimer's disease and related dementias
National Institutes of Health
Grant: 5UL1TR002243-05
Vanderbilt Institute for Clinical and Translational Research (VICTR)
National Institutes of Health
Grant: 5R01GM117650-02
Neurobiology of the Circadian Clock
National Institutes of Health
Grant: 3UL1RR024975-03S4
The Vanderbilt Institute for Clinical and Translational Research (VICTR) UL1
National Institutes of Health
Grant: 1K76AG060001-01A1
Defective lysosomal membrane fission mediates axonal lysosome accumulation in dystrophic neurites in Alzheimer's disease.
Chan Zuckerberg Initiative
U.S. Department of Health & Human Services | National Institutes of Health
FWCI
12.36
Citation Percentile
1.0%
Influential Citations
1
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 7 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 7 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 8 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 8 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 5 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 3 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 6 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 2 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 4 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 4 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 1 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 2 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 1 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 6 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 3 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes
Additional file 5 of mTOR activation induces endolysosomal remodeling and nonclassical secretion of IL-32 via exosomes in inflammatory reactive astrocytes