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Spatially clustered type I interferon responses at injury borderzones

Nature(2024)10.1038/s41586-024-07806-1Source: DataRank Database

Spatially clustered type I interferon responses at injury borderzones is a research paper published in Nature (2024). On theSindex it has a DataRank of 1.3. It has been cited 62 times, with 58 citing works in its 1-hop citation network.

N/A

1.3DataRank · unranked

Scored on demand from live citation data

Deposits data Open Access62 citations · 58 citing works in its network

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Data availability

Repositories this paper deposited data in (declared in PubMed).

GEOGSE268876, GSE176092, GSE269054

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (15 grants)Impact (FWCI 18.44)OA: hybridTopics (109)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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DataRank Breakdown

Base Score 48%Citation Network 52%

Base Score Contribution

0.621

From this paper's citation signal

Citation Network Contribution

0.685

From 32 citing papers with measurable signal

Learn more about DataRank methodology →

Top 5 citers driving the network score

Ranked by each citer's contribution to N(p) — log1p(C_q) divided by its reference count — out of 58 citers.

The heart is put at risk of rupture by cells bordering damaged tissue
Nature20244 citations8 referencesContributes 0.201
Genetic Mapping of Monocyte Fate Decisions Following Myocardial Infarction
bioRxiv (Cold Spring Harbor Laboratory)202311 citations43 referencesContributes 0.058
Spatial Transcriptomics in Human Cardiac Tissue
International Journal of Molecular Sciences202520 citations58 referencesContributes 0.052
Dual-biomimetic Nanodecoys reprogram cardiac macrophages by suppressing STING signaling for heart repair
Journal of Controlled Release20263 citations31 referencesContributes 0.045
Spatiotemporal dynamics of the cardioimmune niche during lesion repair
Nature Cardiovascular Research202511 citations64 referencesContributes 0.039

Authors (18)

David Calcagno ORCID,Justin Yu ORCID,Bin Zhang ORCID,N. Taghdiri,R. Sehgal ORCID

Live enrichment

PubMed:39198639 PMC:PMC11374671 OpenAlex:https://openalex.org/W4401947398

NHLBI NIH HHS

Grant: T32 HL105373

NCATS NIH HHS

Grant: UL1 TR001442

NIH HHS

Grant: DP5 OD031878

NHLBI NIH HHS

Grant: R00 HL129168

NIH HHS

Grant: S10 OD026929

NHLBI NIH HHS

Grant: T32 HL007444

NIAMS NIH HHS

Grant: DP2 AR075321

Fundação para a Ciência e a Tecnologia, I.P.

Grant: PTDC/CCI-BIO/29266/2017

Deep Drug Discovery and Deployment

National Institutes of Health

Grant: 1DP2AR075321-01

Elucidating Cell Communication Networks during Tissue Inflammation, Fibrosis, and Regeneration

National Institutes of Health

Grant: 1S10OD026929-01

Illumina NovaSeq 6000 Sequencing System

National Institutes of Health

Grant: 4R00HL129168-03

Danger Signaling and Spread of Injury after Myocardial Infarction

National Institutes of Health

Grant: 2UL1TR001442-06

UC San Diego Clinical and Translational Research Institute

National Institutes of Health

Grant: 2T32HL105373-06A1

Integrative Bioengineering of Heart, Vessels, and Blood

National Institutes of Health

Grant: 5T32HL007444-39

Training in Cardiovascular Physiology and Pharmacology

National Institutes of Health

Grant: 5DP5OD031878-02

High-throughput imaging of 3D chromatin regulation events in the nervous system

FWCI

18.44

Citation Percentile

1.0%

Citation Trend

2023

2024

2025

2026

hybridLicense: cc-by

journalhttps://www.nature.com/articles/s41586-024-07806-1.pdf

publisherhttps://www.nature.com/articles/s41586-024-07806-1.pdf

publisherhttps://www.nature.com/articles/s41586-024-07806-1

journalhttps://doi.org/10.1038/s41586-024-07806-1

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39198639

Fields of Study

Cardiac Fibrosis and Remodelinginterferon and immune responsesSignaling Pathways in Disease0301 basic medicine0303 health sciences03 medical and health sciencesAnimalsFemaleHumansMaleMiceDendritic CellsEndothelial CellsFibroblastsGene Expression ProfilingImmunity, InnateInterferon Regulatory Factor-3Interferon Type IMacrophagesMice, Inbred C57BLMyocardial InfarctionMyocytes, CardiacNeutrophilsReceptors, CCR2

MeSH Terms

AnimalsDendritic CellsFemaleFibroblastsHumansImmunity, InnateInterferon Type IMacrophagesMaleMice, Inbred C57BLMyocardial InfarctionNeutrophilsGene Expression ProfilingMyocytes, CardiacEndothelial CellsInterferon Regulatory Factor-3MiceReceptors, CCR2

Keywords

InflammationInterferonMyocardial infarctionMyeloidMyeloid cellsInterferon type IBiologyMedicineImmunologyInternal medicineMaleInbred C57BL (mesh)NeutrophilsFibroblasts (mesh)General Science & Technology (science-metrix)Interferon Type I (mesh)CardiovascularInbred C57BLMiceReceptors2.1 Biological and endogenous factorsInnateAnimals (mesh)Myocytes, Cardiac32 Biomedical and Clinical Sciences (for-2020)Male (mesh)Humans (mesh)Mice (mesh)Interferon Regulatory Factor-3 (mesh)Heart DiseaseCardiovascular (rcdc)FemaleCardiacMyocardial Infarction (mesh)Neutrophils (mesh)CCR2 (mesh)General Science & TechnologyReceptors, CCR2Heart Disease (rcdc)610Heart Disease - Coronary Heart Disease (rcdc)ArticleAnimalsHumans3204 Immunology (for-2020)Heart Disease - Coronary Heart DiseaseMyocytesCardiovascular (hrcs-hc)Biomedical and Clinical SciencesGene Expression ProfilingMacrophagesImmunityEndothelial Cells2.1 Biological and endogenous factors (hrcs-rac)Dendritic CellsFibroblastsGene Expression Profiling (mesh)Cardiac (mesh)Immunity, InnateMice, Inbred C57BLDendritic Cells (mesh)Female (mesh)Macrophages (mesh)Endothelial Cells (mesh)CCR2Innate (mesh)Interferon Regulatory Factor-3

Sustainable Development Goals

SDG 0: Good health and well-being