Extrachromosomal DNA is associated with oncogene amplification and poor outcome across multiple cancers is a research paper published in Nature Genetics (2020). On theSindex it has a DataRank of 9.9. It has been cited 641 times, with 200 citing works in its 1-hop citation network.
Scored on demand from live citation data
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DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
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Base Score Contribution
0.970
From this paper's citation signal
Citation Network Contribution
9.0
From 200 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 200 citers.
U.S. Department of Health & Human Services | NIH | National Cancer Institute
Grant: R01 CA190121
U.S. Department of Health & Human Services | NIH | National Cancer Institute
Grant: R01 CA237208
U.S. Department of Health & Human Services | NIH | National Cancer Institute
Grant: P30 CA034196
U.S. Department of Health & Human Services | National Institutes of Health
Grant: R21 NS114873
U.S. Department of Health & Human Services | National Institutes of Health
Grant: R35CA209919
U.S. Department of Health & Human Services | National Institutes of Health
Grant: GM114362
U.S. Department of Health & Human Services | National Institutes of Health
Grant: NS73831
U.S. Department of Health & Human Services | NIH | National Human Genome Research Institute
Grant: RM1-HG007735
Additional file 10 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 10 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 11 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 11 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 12 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 12 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 5 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 5 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 9 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 9 of Hard wiring of normal tissue-specific chromosome-wide gene expression levels is an additional factor driving cancer type-specific aneuploidies
Additional file 1 of Somatic mutation landscape in a cohort of meningiomas that have undergone grade progression
Additional file 1 of Somatic mutation landscape in a cohort of meningiomas that have undergone grade progression
Additional file 3 of Somatic mutation landscape in a cohort of meningiomas that have undergone grade progression
Additional file 3 of Somatic mutation landscape in a cohort of meningiomas that have undergone grade progression
Additional file 1 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS
Additional file 1 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS
Additional file 2 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS
Additional file 2 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS
Additional file 3 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS
Additional file 3 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS