Profibrotic monocyte-derived alveolar macrophages are expanded in patients with persistent respiratory symptoms and radiographic abnormalities after COVID-19 is a research paper published in Nature Immunology (2024). On theSindex it has a DataRank of 0. It has been cited 55 times.
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American Heart Association (American Heart Association, Inc.)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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National Science Foundation (NSF)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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U.S. Department of Health & Human Services | National Institutes of Health (NIH)
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NLM NIH HHS
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CSRD VA
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National Institutes of Health
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Distinct and Overlapping Pathways of Fibrosis and Emphysema in Cigarette Smokers
National Institutes of Health
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Midwest Murine-Tissue Mapping Center (MM-TMC)
National Institutes of Health
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Mitochondrial maladaptive response of the lung epithelium to elevated CO2 levels
National Institutes of Health
Grant: 9I01CX001777-08
Assessing the role of metabolism in monocyte to macrophage differentiation in pulmonary fibrosis
National Institutes of Health
Grant: 5R01HL153312-04
Lung transplant injury drives chronic lung allograft dysfunction via recruitment ofmonocyte-derived alveolar macrophages
National Institutes of Health
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Novel role for CD163 in ozone induced alterations of pulmonary immunity
National Science Foundation
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NSF-Simons Center for Quantitative Biology
National Institutes of Health
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The Robert H.Lurie Comprehensive Cancer Center
National Institutes of Health
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Northwestern University Clinical and Translational Science Institute (NUCATS)
National Institutes of Health
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FACSAria SORP Cell Sorter
National Institutes of Health
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Targeting abnormal alveolar immune activation and failed epithelial repair in COVID-19
National Institutes of Health
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Microglia mediate cognitive dysfunction in elderly survivors of pneumonia
National Institutes of Health
Grant: 1R01AG068579-01
Reconstructing the temporal landscape of gene regulation in aging
National Institutes of Health
Grant: 5R01ES034350-04
Monocyte-derived alveolar macrophage drives inflammatory response to lung ozone exposure
National Institutes of Health
Grant: 5R01HL116931-05
Airway Inspector: a chest imaging biomarker software platform for COPD
National Institutes of Health
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Dysfunction of microglial proteostasis drives neuroinflammation and cognitive decline following pneumonia
National Institutes of Health
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CT and CXR Phenotyping Platform for Assessing COVID-19 Susceptibility and Severity
National Institutes of Health
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Pathogenesis of lung injury mediated by lung-restricted antibodies
National Institutes of Health
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CXCL10/CXCR3 regulation of ozone-induced epithelial permeability
National Institutes of Health
Grant: 5P01AG049665-05
Disordered Proteostasis as a Driver of Disease in the Aging Lung
National Institutes of Health
Grant: 5R01HL145478-07
Donor nonclassical monocytes mediate primary graft dysfunction and remote organ injury following lung transplant surgery
National Institutes of Health
Grant: 5R01HL147575-04
Role of Spleen educated monocytes in mediating ischemia-reperfusion injury following lung transplant
National Institutes of Health
Grant: 1P01HL154998-01A1
Mechanisms of Recovery from Viral Pneumonia
National Institutes of Health
Grant: 5U19AI135964-02
Successful Clinical Response In Pneumonia Therapy (SCRIPT) Systems Biology Center
National Institutes of Health
Grant: 5T32HL076139-04
Training Program in Lung Biology
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