Ik2/TBK1 and Hook/Dynein, an adaptor complex for early endosome transport, are genetic modifiers of FTD-associated mutant CHMP2B toxicity in Drosophila is a research paper published in Scientific Reports (2020). On theSindex it has a DataRank of 0.683. It has been cited 17 times, with 10 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?
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Base Score Contribution
0.434
From this paper's citation signal
Citation Network Contribution
0.250
From 10 citing papers with measurable signal
Ranked by each citer's contribution to N(p) — log1p(Cq) divided by its reference count — out of 10 citers.
Alzheimer's Society
Grant: AS-JF-16b-004
Alzheimer's Society
Grant: AS-PG-2013-005
Medical Research Council
Grant: MR/M013596/1
Interaction of Rab8 with OCRL1: Synaptic growth function in Frontotemporal Dementia and the Neurodevelopmental Disorder Lowe Syndrome
National Institutes of Health
Grant: R37NS057553
National Institutes of Health
Grant: R01NS101986
National Institutes of Health
Grant: R01NS093097
Alzheimer's Society
Grant: 004
NINDS NIH HHS
Grant: RF1 NS101986
NINDS NIH HHS
Grant: R01 NS066586
NINDS NIH HHS
Grant: R01 NS057553
National Institutes of Health
Grant: 5R01NS093097-05
Prefrontal AMPA receptors in FTD Pathogenesis
National Institutes of Health
Grant: 5R37NS057553-14
Investigating Pathogenic Mechanisms of Frontotemporal Dementia Caused by Mutations in CHMP2B and TBK1
National Institutes of Health
Grant: 5R01NS101986-04
Induced Pluripotent Stem Cells and Drosophila Models of C9ORF72-Related FTD/ALS
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 2 of The ESCRT-III protein VPS4, but not CHMP4B or CHMP2B, is pathologically increased in familial and sporadic ALS neuronal nuclei
Additional file 2 of The ESCRT-III protein VPS4, but not CHMP4B or CHMP2B, is pathologically increased in familial and sporadic ALS neuronal nuclei
Additional file 1 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 1 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 2 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 3 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 2 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 3 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 4 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias
Additional file 4 of The enhanced association between mutant CHMP2B and spastin is a novel pathological link between frontotemporal dementia and hereditary spastic paraplegias