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Proapoptotic Bid is required for pulmonary fibrosis

Proceedings of the National Academy of Sciences(2006)10.1073/pnas.0507604103Source: DataRank Database

Proapoptotic Bid is required for pulmonary fibrosis is a research paper published in Proceedings of the National Academy of Sciences (2006). On theSindex it has a DataRank of 4.5. It has been cited 118 times, with 73 citing works in its 1-hop citation network.

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4.5DataRank · unranked

Scored on demand from live citation data

118 citations · 73 citing works in its network

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Linked data & code

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (5 grants)Impact (FWCI 6.17)OA: bronzeTopics (46)Linked Datasets (18)DataCite Reuse (18)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (0/2)

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

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DataRank Breakdown

Base Score 16%Citation Network 84%

Base Score Contribution

0.717

From this paper's citation signal

Citation Network Contribution

3.8

From 72 citing papers with measurable signal

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Top 5 citers driving the network score

Ranked by each citer's contribution to N(p) — log1p(C_q) divided by its reference count — out of 73 citers.

Epithelial fibroblast triggering and interactions in pulmonary fibrosis
European Respiratory Review200847 citations21 referencesContributes 0.184
Targeted Injury of Type II Alveolar Epithelial Cells Induces Pulmonary Fibrosis
American Journal of Respiratory and Critical Care Medicine2009493 citations38 referencesContributes 0.163
Angiotensin signalling in pulmonary fibrosis
The International Journal of Biochemistry & Cell Biology2011132 citations33 referencesContributes 0.148
Epithelial Cell Apoptosis Causes Acute Lung Injury Masquerading as Emphysema
American Journal of Respiratory Cell and Molecular Biology200968 citations30 referencesContributes 0.141
Abrogation of ER stress-induced apoptosis of alveolar epithelial cells by angiotensin 1–7
American Journal of Physiology-Lung Cellular and Molecular Physiology201349 citations31 referencesContributes 0.126

Authors (11)

Gökhan M. Mutlu ORCID,James Eisenbart,Alyson C. Fuller,Amy A. Bellmeyer,Christina M. Baker

Live enrichment

PubMed:16537427 PMC:PMC1401229 OpenAlex:https://openalex.org/W2121729413

NHLBI NIH HHS

Grant: 1P01 HL 071643-01A1

NHLBI NIH HHS

Grant: 5K08 HL 067835-05

NIGMS NIH HHS

Grant: R01 GM060472

NHLBI NIH HHS

Grant: K08 HL067835

NIGMS NIH HHS

Grant: GM 60472-07

FWCI

6.17

Citation Percentile

1.0%

Citation Trend

2012

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2025

bronze

journalhttps://www.pnas.org/doi/pdf/10.1073/pnas.0507604103

publisherhttps://www.pnas.org/doi/pdf/10.1073/pnas.0507604103

publisherhttps://pnas.org/doi/pdf/10.1073/pnas.0507604103

journalhttps://doi.org/10.1073/pnas.0507604103

repositoryhttps://pubmed.ncbi.nlm.nih.gov/16537427

Fields of Study

Interstitial Lung Diseases and Idiopathic Pulmonary FibrosisInhalation and Respiratory Drug DeliveryCardiac electrophysiology and arrhythmiasAnimalsApoptosisBH3 Interacting Domain Death Agonist ProteinBleomycinBronchoalveolar Lavage FluidEpithelial CellsFibroblastsLungMiceMice, Mutant StrainsPulmonary FibrosisTransforming Growth Factor betaTransforming Growth Factor beta1

MeSH Terms

AnimalsBleomycinBronchoalveolar Lavage FluidEpithelial CellsFibroblastsLungMice, Mutant StrainsPulmonary FibrosisTransforming Growth Factor betaApoptosisBH3 Interacting Domain Death Agonist ProteinMiceTransforming Growth Factor beta1

Keywords

BleomycinPulmonary fibrosisFibrosisLungBronchoalveolar lavageMedicineTransforming growth factorIdiopathic pulmonary fibrosisPathologyApoptosisInflammationImmunologyCancer researchInternal medicineBiologyChemotherapyBiochemistry

Sustainable Development Goals

SDG 0: Good health and well-being

Additional file 1 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 1 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 2 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 2 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 3 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 3 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 4 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 4 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 5 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 5 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

10.6084/m9.figshare.22621489figshare

Additional file 6 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 6 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 7 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 7 of UGRP1-modulated MARCO+ alveolar macrophages contribute to age-related lung fibrosis

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Additional file 2 of The HDL from septic-ARDS patients with composition changes exacerbates pulmonary endothelial dysfunction and acute lung injury induced by cecal ligation and puncture (CLP) in mice

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Additional file 1 of The HDL from septic-ARDS patients with composition changes exacerbates pulmonary endothelial dysfunction and acute lung injury induced by cecal ligation and puncture (CLP) in mice

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10.6084/m9.figshare.13192468.v1figshare

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