Glycoprotein nonmetastatic melanoma protein B: A key mediator and an emerging therapeutic target in autoimmune diseases is a research paper published in The FASEB Journal (2020). On theSindex it has a DataRank of 0. It has been cited 57 times.
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NIAID NIH HHS
Grant: U19 AI110502
NIAID NIH HHS
Grant: R01 AI097134
NIAMS NIH HHS
Grant: R01 AR070148
National Institutes of Health
Grant: 1R01AR070148-01A1
Characterizing the Takayasu Arteritis Genetic Risk in RPS9/LILRB3
FWCI
2.32
Citation Percentile
0.9%
Influential Citations
2
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Single-cell transcriptome sequencing–based analysis: probing the mechanisms of glycoprotein NMB regulation of epithelial cells involved in silicosis
Additional file 1 of Single-cell transcriptome sequencing–based analysis: probing the mechanisms of glycoprotein NMB regulation of epithelial cells involved in silicosis
Additional file 2 of Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutations
Additional file 2 of Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutations
Additional file 3 of Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutations
Additional file 3 of Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutations
Additional file 2 of Single-cell transcriptome sequencing–based analysis: probing the mechanisms of glycoprotein NMB regulation of epithelial cells involved in silicosis
Additional file 2 of Single-cell transcriptome sequencing–based analysis: probing the mechanisms of glycoprotein NMB regulation of epithelial cells involved in silicosis