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A natural variation-based screen in mouse cells reveals USF2 as a regulator of the DNA damage response and cellular senescence

bioRxiv (Cold Spring Harbor Laboratory)(2022)10.1101/2022.04.21.489100Source: DataRank Database

A natural variation-based screen in mouse cells reveals USF2 as a regulator of the DNA damage response and cellular senescence is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2022). On theSindex it has a DataRank of 0.

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (4 grants)OA: greenTopics (35)IDs (PubMed, OpenAlex)SDGs (1)

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Authors (8)

Emily C. Moore ORCID,Emily E. K. Kopania ORCID,Christina D. King ORCID,Birgit Schilling ORCID,Judith Campisi ORCID

Live enrichment

PubMed:19965766 OpenAlex:https://openalex.org/W4280634607

National Science Foundation

Grant: 1313190

Graduate Research Fellowship Program (GRFP)

National Institutes of Health

Grant: 5R01HD094787-02

The evolution of genomic imprinting

National Institutes of Health

Grant: 3R01GM120430-06S2

Mapping deep evolutionary divergences in cellular models of stress response

National Institutes of Health

Grant: 7R01NS116992-02

Genetic dissection of trait variation between long-diverged mouse species

greenLicense: cc-by-nc-nd

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2023/01/23/2022.04.21.489100.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2022.04.21.489100

repositoryhttps://doi.org/10.1101/2022.04.21.489100

https://doi.org/10.1093/g3journal/jkad091

Fields of Study

Telomeres, Telomerase, and SenescenceGenomics and Chromatin DynamicsCRISPR and Genetic Engineering0301 basic medicine0303 health sciences03 medical and health sciences

Keywords

SenescenceBiologyDNA damageDNA repairCell cycle checkpointCell biologyCell cycleTranscription factorCellGeneticsGeneDNAInvestigation570610USF2novel screenMice5.1 Pharmaceuticalscellular senescence2.1 Biological and endogenous factorsAnimalsCytokinesUpstream Stimulatory Factorsnatural variationGeneric health relevanceDevelopment of treatments and therapeutic interventionsAetiologyTumor Suppressor Protein p53

Sustainable Development Goals

SDG 0: Life in Land