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Bacterial strain-dependent dissociation of cell recruitment and cell-to-cell spread in early M. tuberculosis infection

bioRxiv (Cold Spring Harbor Laboratory)(2022)10.1101/2022.05.10.491435Source: DataRank Database

Bacterial strain-dependent dissociation of cell recruitment and cell-to-cell spread in early M. tuberculosis infection is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2022). On theSindex it has a DataRank of 0.

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (4 grants)OA: greenTopics (53)IDs (PubMed, OpenAlex)SDGs (2)

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Authors (7)

Ludovic Desvignes ORCID,Tawania Fergus,Amber Cornelius,Tan‐Yun Cheng ORCID,D. Branch Moody ORCID

Live enrichment

PubMed:21097893 OpenAlex:https://openalex.org/W4280621096

National Institutes of Health

Grant: 5K08HL163405-02

Diversity and function of mononuclear phagocytes in the lung driven by mycobacterium tuberculosis infection

National Institutes of Health

Grant: 5P30CA016087-32

Experimental Pathology

National Institutes of Health

Grant: 5R01AI049313-25

T cell responses to CD1-restricted lipids in tuberculosis

National Institutes of Health

Grant: 7R01AI051242-16

Cellular and Bacterial Dynamics in Tuberculosis

greenLicense: cc-by-nc-nd

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2022/05/12/2022.05.10.491435.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2022.05.10.491435

repositoryhttps://doi.org/10.1101/2022.05.10.491435

https://doi.org/10.1128/mbio.01332-22

Fields of Study

Tuberculosis Research and EpidemiologyMycobacterium research and diagnosisImmune responses and vaccinations0301 basic medicine03 medical and health sciences0303 health sciences

Keywords

BiologyTuberculosisMycobacterium tuberculosisImmune systemImmunologyMonocyteInnate immune systemCellPopulationMicrobiologyAcquired immune systemMedicinePathologyGenetics610macrophageAlveolarVaccine RelatedMiceRare DiseasesBiodefense616Macrophages, Alveolar2.2 Factors relating to the physical environment2.1 Biological and endogenous factorsInnateAnimalsdendritic cellsAetiologyinnate immunityLungT cell activationPreventionInflammatory and immune systemMacrophagesImmunityQR1-502Immunity, InnateT cell primingInfectious DiseasesEmerging Infectious DiseasesGood Health and Well Beinginnate responsestrain diversityInfectionBeijing strainResearch Article

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being