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MYC acetylated lysine residues drive oncogenic cell transformation and regulate select genetic programs for cell adhesion-independent growth and survival

Genes & Development(2023)10.1101/gad.350736.123Source: DataRank Database

MYC acetylated lysine residues drive oncogenic cell transformation and regulate select genetic programs for cell adhesion-independent growth and survival is a research paper published in Genes & Development (2023). On theSindex it has a DataRank of 0. It has been cited 16 times.

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Enrichment:Funding (21 grants)OA: goldTopics (69)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

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Authors (18)

Jeffrey J. Pino,Kay Jang,Michael M. Allevato ORCID,Marina Vorontchikhina,Wataru Ichikawa ORCID

Live enrichment

PubMed:37852796 PMC:PMC10691474 OpenAlex:https://openalex.org/W4387736539

National Institutes of Health

Grant: 1F31CA165776

National Institutes of Health

Grant: T34GM062756

U.S Department of Education

Grant: P200A210136

National Cancer Institute

Grant: R01CA158540

National Cancer Institute

Grant: R03CA230843

National Cancer Institute

Grant: R03CA249489

National Cancer Institute

Grant: P20CA242620

National Cancer Institute

Grant: P20CA242619

NIGMS NIH HHS

Grant: T34 GM149470

NCI NIH HHS

Grant: U54 CA285116

NCI NIH HHS

Grant: F31 CA165776

NCI NIH HHS

Grant: U54 CA285114

National Institutes of Health

Grant: 5F31CA165776-02

Functional Definition of MYC Acetylation

National Institutes of Health

Grant: 1P20CA242620-01

2/2 Drug Development and Capacity Building: A UCR/CoH-CCC Partnership

National Institutes of Health

Grant: 5P20CA242619-04

1/2 Drug Development and Capacity Building: A UCR/CoH-CCC Partnership

National Institutes of Health

Grant: 5R01CA158540-02

Functional characterization of the long non-coding RNA Scirocco in development an

National Institutes of Health

Grant: 5R03CA249489-02

Role of MYC Acetylation in Oncogenic Transformation

National Institutes of Health

Grant: 5T34GM062756-08

UC Riverside MARC U* Star Undergraduate Research Program

National Institutes of Health

Grant: 1R03CA230843-01A1

Role of MYC Acetylation in Cancer

National Cancer Institute

National Institute of General Medical Sciences

goldLicense: cc-by-nc

journalhttp://genesdev.cshlp.org/content/early/2023/10/18/gad.350736.123.full.pdf

GOLDhttps://doi.org/10.1101/gad.350736.123

publisherhttp://genesdev.cshlp.org/content/early/2023/10/18/gad.350736.123.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/gad.350736.123

repositoryhttps://pubmed.ncbi.nlm.nih.gov/37852796

Fields of Study

Histone Deacetylase Inhibitors ResearchUbiquitin and proteasome pathwaysPeptidase Inhibition and AnalysisMedicineBiology0301 basic medicine03 medical and health sciencesAnimalsHumansMiceAcetylationCell AdhesionCell ProliferationCell Transformation, NeoplasticHistone AcetyltransferasesLysineProto-Oncogene Proteins c-myc

MeSH Terms

AcetylationAnimalsCell AdhesionCell Transformation, NeoplasticHumansLysineProto-Oncogene Proteins c-mycCell ProliferationMiceHistone Acetyltransferases

Keywords

AcetylationBiologyLysineHDAC8Cell growthTranscription factorP300-CBP Transcription FactorsCell biologyHistone deacetylaseHistoneCancer researchBiochemistryGeneHistone AcetyltransferasesAmino acidHistone H2AMalignant transformationLysine AcetylationMyc OncoproteinBiomedical and clinical sciences1.1 Normal biological development and functioning610Cell TransformationMedical and Health SciencesProto-Oncogene Proteins c-mycMiceGeneticsCell Adhesion2.1 Biological and endogenous factorsPsychologyAnimalsHumansCancerCell ProliferationNeoplasticPsychology and Cognitive SciencesBiological Sciences540Research PapersCell Transformation, NeoplasticBiochemistry and Cell BiologyDevelopmental Biology

Sustainable Development Goals

SDG 0: Good health and well-being