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Mitochondrial protein adduct and superoxide generation are prerequisites for early activation of c-jun N-terminal kinase within the cytosol after an acetaminophen overdose in mice

Toxicology Letters(2020)10.1016/j.toxlet.2020.12.005Source: DataRank Database

Mitochondrial protein adduct and superoxide generation are prerequisites for early activation of c-jun N-terminal kinase within the cytosol after an acetaminophen overdose in mice is a research paper published in Toxicology Letters (2020). On theSindex it has a DataRank of 0. It has been cited 68 times.

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Open Access68 citations

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Enrichment:Funding (7 grants)Impact (FWCI 5.91)OA: greenTopics (66)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (1)

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Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (6)

Kuo Du ORCID,Jephte Y. Akakpo ORCID,David S. Umbaugh ORCID,Hartmut Jaeschke ORCID,Anup Ramachandran ORCID

Live enrichment

PubMed:33290831 PMC:PMC7852579 OpenAlex:https://openalex.org/W3112445434

NIGMS NIH HHS

Grant: P30 GM118247

NIGMS NIH HHS

Grant: P20 GM103549

NIDDK NIH HHS

Grant: R01 DK102142

National Institutes of Health

Grant: 5R01DK102142-07

Autophagy and Drug-Induced Liver Injury

National Institutes of Health

Grant: 5P20GM103549-08

Nuclear Receptors in Liver Health and Disease

National Institutes of Health

Grant: 5P30GM118247-05

Mechanisms of Liver Injury and Diseases

National Institutes of Health

FWCI

5.91

Citation Percentile

1.0%

Influential Citations

Citation Trend

2020

2021

2022

2023

2024

2025

2026

greenLicense: Elsevier TDM

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7852579

GREENhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC7852579

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7852579

publisherhttps://api.elsevier.com/content/article/PII:S0378427420304987?httpAccept=text/xml

publisherhttps://api.elsevier.com/content/article/PII:S0378427420304987?httpAccept=text/plain

Fields of Study

Drug-Induced Hepatotoxicity and ProtectionMalaria Research and ControlMetabolomics and Mass Spectrometry StudiesChemistryMedicine0301 basic medicine0303 health sciences03 medical and health sciences

MeSH Terms

AcetaminophenAnimalsCells, CulturedCytosolDisease Models, AnimalEnzyme ActivationIminesMaleMice, Inbred C57BLMitochondria, LiverSuperoxidesTime FactorsBenzoquinonesOxidative StressProtein TransportHepatocytesMitochondrial ProteinsJNK Mitogen-Activated Protein KinasesMiceChemical and Drug Induced Liver InjuryDrug Overdose

Keywords

CytosolTerminal (telecommunication)AdductChemistrySuperoxidec-junMitochondrionKinaseAcetaminophenBiochemistryEnzymeGeneComputer networkComputer scienceOrganic chemistryMitochondriaOxidative stressElectron Transport ChainNapqiProtein AdductsMaleTime FactorsJNK Mitogen-Activated Protein KinasesMitochondria, LiverEnzyme ActivationMice, Inbred C57BLMitochondrial ProteinsDisease Models, AnimalProtein TransportSuperoxidesBenzoquinonesHepatocytesAnimalsIminesChemical and Drug Induced Liver InjuryDrug OverdoseCells, Cultured

Sustainable Development Goals

SDG 0: Good health and well-being

Mitochondrial protein adduct and superoxide generation are prerequisites for early activation of c-jun N-terminal kinase within the cytosol after an acetaminophen overdose in mice

FAIR Checklist

Authors (6)

Funding (7 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)