🏆 Finalist — NIH Data Sharing Index (“S-Index”) Challenge

Fostering Data Sharing

Search Papers Authors Institutions Grants FAIR About

TheSindex

Fostering Data Sharing

Empowering the data sharing that empowers science — an open, citation-grounded index for datasets.

Product

Search
API
Explore
Rankings

Resources

Methodology
Data Sources
Documentation
Press

Organization

About
Team
Wikidata

© 2026 theSindex. All rights reserved.

Home
Papers
Targeted Mutational Analysis of Cortisol-Producing Adenomas

Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Targeted Mutational Analysis of Cortisol-Producing Adenomas

The Journal of Clinical Endocrinology & Metabolism(2021)10.1210/clinem/dgab682Source: DataRank Database

Targeted Mutational Analysis of Cortisol-Producing Adenomas is a research paper published in The Journal of Clinical Endocrinology & Metabolism (2021). On theSindex it has a DataRank of 0. It has been cited 33 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access33 citations

Cite:

Linked data & code

gen refseq

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (23 grants)OA: hybridTopics (44)IDs (PubMed, PMC)Annotations (2)SDGs (1)

FAIR Checklist

Context only (not used in score)

FFindable (1/2)

Has DOI

AAccessible (1/2)

Open Access

IInteroperable (0/2)

RReusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (12)

Jessie Hoxie,Chia-Jen Liu ORCID,Morgan N Cash,James M. Luther ORCID,Lan L. Gellert ORCID

Live enrichment

PubMed:34534321 PMC:PMC8764218

National Institute of Diabetes and Digestive and Kidney Diseases

Grant: R01DK106618

National Institute of Diabetes and Digestive and Kidney Diseases

Grant: R01DK043140

National Institute of Diabetes and Digestive and Kidney Diseases

Grant: 1K08DK109116

National Institute of Diabetes and Digestive and Kidney Diseases

Grant: R01DK096994

National Institute of Diabetes and Digestive and Kidney Diseases

Grant: R01DK081662

National Cancer Institute

Grant: 2P30CA068485

National Center for Advancing Translational Sciences

Grant: UL1TR002243

Michigan Institute for Clinical and Health Research

Grant: UL1TR002240

National Heart, Lung, and Blood Institute

Grant: 1R01HL130106

Doris Duke Charitable Foundation

Grant: 2019087

American Heart Association

Grant: 17SDG33660447

American Heart Association

Grant: 20CDA35320016

NCI NIH HHS

Grant: P30 CA068485

NIDDK NIH HHS

Grant: K08 DK109116

NHLBI NIH HHS

Grant: R01 HL130106

NIDDK NIH HHS

Grant: K23 DK081662

National Institutes of Health

Grant: 5UL1TR002240-02

Michigan Institute for Clinical and Health Research (MICHR)

National Institutes of Health

Grant: 5R01HL130106-02

The Genetics of Primary Aldosteronism

National Institutes of Health

Grant: 5R01DK096994-05

Endogenous Aldosterone and Glucose Homeostasis

National Institutes of Health

Grant: 5UL1TR002243-05

Vanderbilt Institute for Clinical and Translational Research (VICTR)

National Institutes of Health

Grant: 2R01DK043140-06

MECHANISMS LEADING TO ADRENAL ZONATION

National Institutes of Health

Grant: 5K08DK109116-04

The contemporary endocrinology of congenital adrenal hyperplasia

National Institutes of Health

Grant: 5R01DK106618-08

Adrenal Origins of Aldosterone Excess

hybridLicense: OUP Standard Publication Reuse

publisherhttp://academic.oup.com/jcem/advance-article-pdf/doi/10.1210/clinem/dgab682/40539447/dgab682.pdf

publisherhttps://academic.oup.com/jcem/article-pdf/107/2/e594/42224478/dgab682.pdf

https://doi.org/10.1210/clinem/dgab682

https://pubmed.ncbi.nlm.nih.gov/34534321

https://dx.doi.org/10.1210/clinem/dgab682

Fields of Study

03 medical and health sciences0302 clinical medicine

MeSH Terms

Adrenal GlandsHumansAdrenal Cortex NeoplasmsAdrenocortical AdenomaCushing SyndromeHydrocortisoneGTP-Binding Protein alpha Subunits, GsChromograninsAdrenalectomyDNA Mutational AnalysisMutationAdultMiddle AgedFemaleMalebeta CateninCyclic AMP-Dependent Protein Kinase Catalytic SubunitsCyclic AMP-Dependent Protein Kinase RIalpha SubunitPatient Acuity

Keywords

Cushing SyndromeMutational AnalysisTargeted Amplicon SequencingCortisol-producing AdenomaMild Autonomous Cortisol ExcessAdultMaleCyclic AMP-Dependent Protein Kinase Catalytic SubunitsHydrocortisoneCyclic AMP-Dependent Protein Kinase RIalpha SubunitDNA Mutational AnalysisPatient AcuityAdrenalectomyMiddle AgedAdrenal Cortex NeoplasmsAdrenal GlandsAdrenocortical AdenomaMutationChromograninsGTP-Binding Protein alpha Subunits, GsHumansFemalebeta Catenin

Sustainable Development Goals

SDG 3: 3. Good health

Related Papers (10)

Genomic and sequence variants of protein kinase A regulatory subunit type 1β (PRKAR1B) in patients with adrenocortical disease and Cushing syndrome

Genetics in Medicine(2020)

co-cited

10.1038/s41436-020-00958-1

Sleep and circadian regulation of cortisol: A short review

Current Opinion in Endocrine and Metabolic Research(2021)

co-cited

10.1016/j.coemr.2021.03.011

Endosomal cAMP production broadly impacts the cellular phosphoproteome

Journal of Biological Chemistry(2021)

co-cited

10.1016/j.jbc.2021.100907

Variants in PRKAR1B cause a neurodevelopmental disorder with autism spectrum disorder, apraxia, and insensitivity to pain

Genetics in Medicine(2021)

co-cited

10.1038/s41436-021-01152-7

Is Disrupted Nucleotide-Substrate Cooperativity a Common Trait for Cushing's Syndrome Driving Mutations of Protein Kinase A?

Journal of Molecular Biology(2021)

co-cited

10.1016/j.jmb.2021.167123

A PKA inhibitor motif within SMOOTHENED controls Hedgehog signal transduction

Nature Structural & Molecular Biology(2022)

co-cited

10.1038/s41594-022-00838-z

Mislocalization of protein kinase A drives pathology in Cushing’s syndrome

N/A

0.553DataRank · unranked

Cell Reports(2022)

co-cited

10.1016/j.celrep.2022.111073

Oncogenic PKA signaling increases c-MYC protein expression through multiple targetable mechanisms

N/A

0.725DataRank · unranked

eLife(2023)

co-cited

10.7554/elife.69521

Classification of Cushing's syndrome PKAc mutants based upon their ability to bind PKI

Biochemical Journal(2023)

co-cited

10.1042/bcj20230183

Dark kinase annotation, mining, and visualization using the Protein Kinase Ontology

Top 36%

0.538DataRank

PeerJ(2023)

co-cited

10.7717/peerj.16087